Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 453 entries
Sorted by: Best Match Show Resources per page
Association of blood manganese level with diabetes and renal dysfunction: a cross-sectional study of the Korean general population.

BMC endocrine disorders

Koh ES, Kim SJ, Yoon HE, Chung JH, Chung S, Park CW, Chang YS, Shin SJ.
PMID: 24606630
BMC Endocr Disord. 2014 Mar 08;14:24. doi: 10.1186/1472-6823-14-24.

BACKGROUND: The purpose of this study was to evaluate the association between blood manganese levels and the prevalence of chronic diseases in the Korean population.METHODS: This was a cross-sectional study based on the Korean National Health and Nutrition Examination...

Cite
Koh ES, Kim SJ, Yoon HE, et al. Association of blood manganese level with diabetes and renal dysfunction: a cross-sectional study of the Korean general population. BMC Endocr Disord. 2014;14:24doi: 10.1186/1472-6823-14-24.
Koh, E. S., Kim, S. J., Yoon, H. E., Chung, J. H., Chung, S., Park, C. W., Chang, Y. S., & Shin, S. J. (2014). Association of blood manganese level with diabetes and renal dysfunction: a cross-sectional study of the Korean general population. BMC endocrine disorders, 1424. https://doi.org/10.1186/1472-6823-14-24
Koh, Eun Sil, et al. "Association of blood manganese level with diabetes and renal dysfunction: a cross-sectional study of the Korean general population." BMC endocrine disorders vol. 14 (2014): 24. doi: https://doi.org/10.1186/1472-6823-14-24
Koh ES, Kim SJ, Yoon HE, Chung JH, Chung S, Park CW, Chang YS, Shin SJ. Association of blood manganese level with diabetes and renal dysfunction: a cross-sectional study of the Korean general population. BMC Endocr Disord. 2014 Mar 08;14:24. doi: 10.1186/1472-6823-14-24. PMID: 24606630; PMCID: PMC3973834.
Copy Download .nbib Format: NLM
Unilateral adrenal hyperplasia is a usual cause of primary hyperaldosteronism. Results from a Swedish screening study.

BMC endocrine disorders

Sigurjonsdottir HA, Gronowitz M, Andersson O, Eggertsen R, Herlitz H, Sakinis A, Wangberg B, Johannsson G.
PMID: 22958674
BMC Endocr Disord. 2012 Sep 08;12:17. doi: 10.1186/1472-6823-12-17.

BACKGROUND: The existence of unilateral adrenal hyperplasia (AH) has been considered a rare cause of primary hyperaldosteronism (PA).METHODS: In a prospective study we screened for PA in a non-selected (NSP) and selected hypertensive population (SP), to define the cause...

Cite
Sigurjonsdottir HA, Gronowitz M, Andersson O, et al. Unilateral adrenal hyperplasia is a usual cause of primary hyperaldosteronism. Results from a Swedish screening study. BMC Endocr Disord. 2012;12:17doi: 10.1186/1472-6823-12-17.
Sigurjonsdottir, H. A., Gronowitz, M., Andersson, O., Eggertsen, R., Herlitz, H., Sakinis, A., Wangberg, B., & Johannsson, G. (2012). Unilateral adrenal hyperplasia is a usual cause of primary hyperaldosteronism. Results from a Swedish screening study. BMC endocrine disorders, 1217. https://doi.org/10.1186/1472-6823-12-17
Sigurjonsdottir, Helga Agusta, et al. "Unilateral adrenal hyperplasia is a usual cause of primary hyperaldosteronism. Results from a Swedish screening study." BMC endocrine disorders vol. 12 (2012): 17. doi: https://doi.org/10.1186/1472-6823-12-17
Sigurjonsdottir HA, Gronowitz M, Andersson O, Eggertsen R, Herlitz H, Sakinis A, Wangberg B, Johannsson G. Unilateral adrenal hyperplasia is a usual cause of primary hyperaldosteronism. Results from a Swedish screening study. BMC Endocr Disord. 2012 Sep 08;12:17. doi: 10.1186/1472-6823-12-17. PMID: 22958674; PMCID: PMC3515501.
Copy Download .nbib Format: NLM
Genetic analyses of bone morphogenetic protein 2, 4 and 7 in congenital combined pituitary hormone deficiency.

BMC endocrine disorders

Breitfeld J, Martens S, Klammt J, Schlicke M, Pfäffle R, Krause K, Weidle K, Schleinitz D, Stumvoll M, Führer D, Kovacs P, Tönjes A.
PMID: 24289245
BMC Endocr Disord. 2013 Dec 01;13:56. doi: 10.1186/1472-6823-13-56.

BACKGROUND: The complex process of development of the pituitary gland is regulated by a number of signalling molecules and transcription factors. Mutations in these factors have been identified in rare cases of congenital hypopituitarism but for most subjects with...

Cite
Breitfeld J, Martens S, Klammt J, et al. Genetic analyses of bone morphogenetic protein 2, 4 and 7 in congenital combined pituitary hormone deficiency. BMC Endocr Disord. 2013;13:56doi: 10.1186/1472-6823-13-56.
Breitfeld, J., Martens, S., Klammt, J., Schlicke, M., Pfäffle, R., Krause, K., Weidle, K., Schleinitz, D., Stumvoll, M., Führer, D., Kovacs, P., & Tönjes, A. (2013). Genetic analyses of bone morphogenetic protein 2, 4 and 7 in congenital combined pituitary hormone deficiency. BMC endocrine disorders, 1356. https://doi.org/10.1186/1472-6823-13-56
Breitfeld, Jana, et al. "Genetic analyses of bone morphogenetic protein 2, 4 and 7 in congenital combined pituitary hormone deficiency." BMC endocrine disorders vol. 13 (2013): 56. doi: https://doi.org/10.1186/1472-6823-13-56
Breitfeld J, Martens S, Klammt J, Schlicke M, Pfäffle R, Krause K, Weidle K, Schleinitz D, Stumvoll M, Führer D, Kovacs P, Tönjes A. Genetic analyses of bone morphogenetic protein 2, 4 and 7 in congenital combined pituitary hormone deficiency. BMC Endocr Disord. 2013 Dec 01;13:56. doi: 10.1186/1472-6823-13-56. PMID: 24289245; PMCID: PMC4175098.
Copy Download .nbib Format: NLM
Erratum to: Ectopic ACTH secretion associated to a well-differentiated peritoneal mesothelioma.

BMC endocrine disorders

Mendoza CF, Ontiveros P, Xibillé DX, Rivera HM.
PMID: 26370229
BMC Endocr Disord. 2015 Sep 14;15:48. doi: 10.1186/s12902-015-0046-x.

No abstract available.

Cite
Mendoza CF, Ontiveros P, Xibillé DX, et al. Erratum to: Ectopic ACTH secretion associated to a well-differentiated peritoneal mesothelioma. BMC Endocr Disord. 2015;15:48doi: 10.1186/s12902-015-0046-x.
Mendoza, C. F., Ontiveros, P., Xibillé, D. X., & Rivera, H. M. (2015). Erratum to: Ectopic ACTH secretion associated to a well-differentiated peritoneal mesothelioma. BMC endocrine disorders, 1548. https://doi.org/10.1186/s12902-015-0046-x
Mendoza, Carmen F, et al. "Erratum to: Ectopic ACTH secretion associated to a well-differentiated peritoneal mesothelioma." BMC endocrine disorders vol. 15 (2015): 48. doi: https://doi.org/10.1186/s12902-015-0046-x
Mendoza CF, Ontiveros P, Xibillé DX, Rivera HM. Erratum to: Ectopic ACTH secretion associated to a well-differentiated peritoneal mesothelioma. BMC Endocr Disord. 2015 Sep 14;15:48. doi: 10.1186/s12902-015-0046-x. PMID: 26370229; PMCID: PMC4570587.
Copy Download .nbib Format: NLM
Familial multinodular goiter syndrome with papillary thyroid carcinomas: mutational analysis of the associated genes in 5 cases from 1 Chinese family.

BMC endocrine disorders

Liao S, Song W, Liu Y, Deng S, Liang Y, Tang Z, Huang J, Dong D, Xu G.
PMID: 24144365
BMC Endocr Disord. 2013 Oct 21;13:48. doi: 10.1186/1472-6823-13-48.

BACKGROUND: Familial papillary thyroid cancer (fPTC) is recognized as a distinct entity only recently and no fPTC predisposing genes have been identified. Several potential regions and susceptibility loci for sporadic PTC have been reported. We aimed to evaluate the...

Cite
Liao S, Song W, Liu Y, et al. Familial multinodular goiter syndrome with papillary thyroid carcinomas: mutational analysis of the associated genes in 5 cases from 1 Chinese family. BMC Endocr Disord. 2013;13:48doi: 10.1186/1472-6823-13-48.
Liao, S., Song, W., Liu, Y., Deng, S., Liang, Y., Tang, Z., Huang, J., Dong, D., & Xu, G. (2013). Familial multinodular goiter syndrome with papillary thyroid carcinomas: mutational analysis of the associated genes in 5 cases from 1 Chinese family. BMC endocrine disorders, 1348. https://doi.org/10.1186/1472-6823-13-48
Liao, Shunyao, et al. "Familial multinodular goiter syndrome with papillary thyroid carcinomas: mutational analysis of the associated genes in 5 cases from 1 Chinese family." BMC endocrine disorders vol. 13 (2013): 48. doi: https://doi.org/10.1186/1472-6823-13-48
Liao S, Song W, Liu Y, Deng S, Liang Y, Tang Z, Huang J, Dong D, Xu G. Familial multinodular goiter syndrome with papillary thyroid carcinomas: mutational analysis of the associated genes in 5 cases from 1 Chinese family. BMC Endocr Disord. 2013 Oct 21;13:48. doi: 10.1186/1472-6823-13-48. PMID: 24144365; PMCID: PMC4015857.
Copy Download .nbib Format: NLM
Hypertension secondary to a periprostatic paraganglioma: case report and review of the literature.

BMC endocrine disorders

Kers J, Choudhry ZA, Roeleveld TA, Houdijk AP.
PMID: 24274233
BMC Endocr Disord. 2013 Nov 25;13:55. doi: 10.1186/1472-6823-13-55.

BACKGROUND: Around 10 per cent of catecholamine-secreting tumours can be found outside the adrenal medulla (paraganglioma). We report a case of a functional sporadic paraganglioma that was localized lateral to the prostate without causing lower urinary tract symptoms.CASE PRESENTATION:...

Cite
Kers J, Choudhry ZA, Roeleveld TA, et al. Hypertension secondary to a periprostatic paraganglioma: case report and review of the literature. BMC Endocr Disord. 2013;13:55doi: 10.1186/1472-6823-13-55.
Kers, J., Choudhry, Z. A., Roeleveld, T. A., & Houdijk, A. P. (2013). Hypertension secondary to a periprostatic paraganglioma: case report and review of the literature. BMC endocrine disorders, 1355. https://doi.org/10.1186/1472-6823-13-55
Kers, Jesper, et al. "Hypertension secondary to a periprostatic paraganglioma: case report and review of the literature." BMC endocrine disorders vol. 13 (2013): 55. doi: https://doi.org/10.1186/1472-6823-13-55
Kers J, Choudhry ZA, Roeleveld TA, Houdijk AP. Hypertension secondary to a periprostatic paraganglioma: case report and review of the literature. BMC Endocr Disord. 2013 Nov 25;13:55. doi: 10.1186/1472-6823-13-55. PMID: 24274233; PMCID: PMC4175107.
Copy Download .nbib Format: NLM
Different thresholds of tissue-specific dose-responses to growth hormone in short prepubertal children.

BMC endocrine disorders

Decker R, Nygren A, Kriström B, Nierop AF, Gustafsson J, Albertsson-Wikland K, Dahlgren J.
PMID: 23116291
BMC Endocr Disord. 2012 Nov 01;12:26. doi: 10.1186/1472-6823-12-26.

BACKGROUND: In addition to stimulating linear growth in children, growth hormone (GH) influences metabolism and body composition. These effects should be considered when individualizing GH treatment as dose-dependent changes in metabolic markers have been reported.HYPOTHESIS: There are different dose-dependent...

Cite
Decker R, Nygren A, Kriström B, et al. Different thresholds of tissue-specific dose-responses to growth hormone in short prepubertal children. BMC Endocr Disord. 2012;12:26doi: 10.1186/1472-6823-12-26.
Decker, R., Nygren, A., Kriström, B., Nierop, A. F., Gustafsson, J., Albertsson-Wikland, K., & Dahlgren, J. (2012). Different thresholds of tissue-specific dose-responses to growth hormone in short prepubertal children. BMC endocrine disorders, 1226. https://doi.org/10.1186/1472-6823-12-26
Decker, Ralph, et al. "Different thresholds of tissue-specific dose-responses to growth hormone in short prepubertal children." BMC endocrine disorders vol. 12 (2012): 26. doi: https://doi.org/10.1186/1472-6823-12-26
Decker R, Nygren A, Kriström B, Nierop AF, Gustafsson J, Albertsson-Wikland K, Dahlgren J. Different thresholds of tissue-specific dose-responses to growth hormone in short prepubertal children. BMC Endocr Disord. 2012 Nov 01;12:26. doi: 10.1186/1472-6823-12-26. PMID: 23116291; PMCID: PMC3583138.
Copy Download .nbib Format: NLM
A case of masked toxic adenoma in a patient with non-thyroidal illness.

BMC endocrine disorders

Cho EA, Yoon JH, Kim HK, Kang HC.
PMID: 24383500
BMC Endocr Disord. 2014 Jan 02;14:1. doi: 10.1186/1472-6823-14-1.

BACKGROUND: Non-thyroidal illness (NTI) refers to changes in thyroid hormone levels in critically ill patients in the absence of primary hypothalamic-pituitary-thyroid dysfunction, and these abnormalities usually resolve after clinical recovery. However, NTI can be accompanied by primary thyroid dysfunction....

Cite
Cho EA, Yoon JH, Kim HK, et al. A case of masked toxic adenoma in a patient with non-thyroidal illness. BMC Endocr Disord. 2014;14:1doi: 10.1186/1472-6823-14-1.
Cho, E. A., Yoon, J. H., Kim, H. K., & Kang, H. C. (2014). A case of masked toxic adenoma in a patient with non-thyroidal illness. BMC endocrine disorders, 141. https://doi.org/10.1186/1472-6823-14-1
Cho, Eun Ae, et al. "A case of masked toxic adenoma in a patient with non-thyroidal illness." BMC endocrine disorders vol. 14 (2014): 1. doi: https://doi.org/10.1186/1472-6823-14-1
Cho EA, Yoon JH, Kim HK, Kang HC. A case of masked toxic adenoma in a patient with non-thyroidal illness. BMC Endocr Disord. 2014 Jan 02;14:1. doi: 10.1186/1472-6823-14-1. PMID: 24383500; PMCID: PMC3883471.
Copy Download .nbib Format: NLM
Gene expression of sternohyoid and diaphragm muscles in type 2 diabetic rats.

BMC endocrine disorders

van Lunteren E, Moyer M.
PMID: 24199937
BMC Endocr Disord. 2013 Oct 07;13:43. doi: 10.1186/1472-6823-13-43.

BACKGROUND: Type 2 diabetes differs from type 1 diabetes in its pathogenesis. Type 1 diabetic diaphragm has altered gene expression which includes lipid and carbohydrate metabolism, ubiquitination and oxidoreductase activity. The objectives of the present study were to assess...

Cite
van Lunteren E, Moyer M. Gene expression of sternohyoid and diaphragm muscles in type 2 diabetic rats. BMC Endocr Disord. 2013;13:43doi: 10.1186/1472-6823-13-43.
van Lunteren, E., & Moyer, M. (2013). Gene expression of sternohyoid and diaphragm muscles in type 2 diabetic rats. BMC endocrine disorders, 1343. https://doi.org/10.1186/1472-6823-13-43
van Lunteren, Erik, and Moyer, Michelle. "Gene expression of sternohyoid and diaphragm muscles in type 2 diabetic rats." BMC endocrine disorders vol. 13 (2013): 43. doi: https://doi.org/10.1186/1472-6823-13-43
van Lunteren E, Moyer M. Gene expression of sternohyoid and diaphragm muscles in type 2 diabetic rats. BMC Endocr Disord. 2013 Oct 07;13:43. doi: 10.1186/1472-6823-13-43. PMID: 24199937; PMCID: PMC3851765.
Copy Download .nbib Format: NLM
Medication utilization patterns among type 2 diabetes patients initiating Exenatide BID or insulin glargine: a retrospective database study.

BMC endocrine disorders

Pawaskar M, Bonafede M, Johnson B, Fowler R, Lenhart G, Hoogwerf B.
PMID: 23799930
BMC Endocr Disord. 2013 Jun 22;13:20. doi: 10.1186/1472-6823-13-20.

BACKGROUND: Type 2 diabetes is a common and costly illness, associated with significant morbidity and mortality. Despite this, there is relatively little information on the 'real-world' medication utilization patterns for patients with type 2 diabetes initiating exenatide BID or...

Cite
Pawaskar M, Bonafede M, Johnson B, et al. Medication utilization patterns among type 2 diabetes patients initiating Exenatide BID or insulin glargine: a retrospective database study. BMC Endocr Disord. 2013;13:20doi: 10.1186/1472-6823-13-20.
Pawaskar, M., Bonafede, M., Johnson, B., Fowler, R., Lenhart, G., & Hoogwerf, B. (2013). Medication utilization patterns among type 2 diabetes patients initiating Exenatide BID or insulin glargine: a retrospective database study. BMC endocrine disorders, 1320. https://doi.org/10.1186/1472-6823-13-20
Pawaskar, Manjiri, et al. "Medication utilization patterns among type 2 diabetes patients initiating Exenatide BID or insulin glargine: a retrospective database study." BMC endocrine disorders vol. 13 (2013): 20. doi: https://doi.org/10.1186/1472-6823-13-20
Pawaskar M, Bonafede M, Johnson B, Fowler R, Lenhart G, Hoogwerf B. Medication utilization patterns among type 2 diabetes patients initiating Exenatide BID or insulin glargine: a retrospective database study. BMC Endocr Disord. 2013 Jun 22;13:20. doi: 10.1186/1472-6823-13-20. PMID: 23799930; PMCID: PMC3750447.
Copy Download .nbib Format: NLM
Possible modulatory effect of endogenous islet catecholamines on insulin secretion.

BMC endocrine disorders

Borelli MI, Gagliardino JJ.
PMID: 11696250
BMC Endocr Disord. 2001;1(1):1. doi: 10.1186/1472-6823-1-1.

BACKGROUND: The possible participation of endogenous islet catecholamines (CAs) in the control of insulin secretion was tested. METHODS: Glucose-induced insulin secretion was measured in the presence of 3-Iodo-L-Tyrosine (MIT), a specific inhibitor of tyrosine-hydroxylase activity, in fresh and precultured...

Cite
Borelli MI, Gagliardino JJ. Possible modulatory effect of endogenous islet catecholamines on insulin secretion. BMC Endocr Disord. 2001;1(1):1doi: 10.1186/1472-6823-1-1.
Borelli, M. I., & Gagliardino, J. J. (2001). Possible modulatory effect of endogenous islet catecholamines on insulin secretion. BMC endocrine disorders, 1(1), 1. https://doi.org/10.1186/1472-6823-1-1
Borelli, Maria I, and Gagliardino, Juan J. "Possible modulatory effect of endogenous islet catecholamines on insulin secretion." BMC endocrine disorders vol. 1,1 (2001): 1. doi: https://doi.org/10.1186/1472-6823-1-1
Borelli MI, Gagliardino JJ. Possible modulatory effect of endogenous islet catecholamines on insulin secretion. BMC Endocr Disord. 2001;1(1):1. doi: 10.1186/1472-6823-1-1. PMID: 11696250; PMCID: PMC59668.
Copy Download .nbib Format: NLM
Association of angiotensin-converting enzyme inhibitor therapy and comorbidity in diabetes: results from the Vermont diabetes information system.

BMC endocrine disorders

Ramos-Nino ME, Maclean CD, Littenberg B.
PMID: 19061507
BMC Endocr Disord. 2008 Dec 05;8:17. doi: 10.1186/1472-6823-8-17.

BACKGROUND: Angiotensin converting enzyme inhibitors (ACE inhibitors) reduce peripheral vascular resistance via blockage of angiotensin converting enzyme (ACE). ACE inhibitors are commonly used to treat congestive heart failure and high blood pressure, but other effects have been reported. In...

Cite
Ramos-Nino ME, Maclean CD, Littenberg B. Association of angiotensin-converting enzyme inhibitor therapy and comorbidity in diabetes: results from the Vermont diabetes information system. BMC Endocr Disord. 2008;8:17doi: 10.1186/1472-6823-8-17.
Ramos-Nino, M. E., Maclean, C. D., & Littenberg, B. (2008). Association of angiotensin-converting enzyme inhibitor therapy and comorbidity in diabetes: results from the Vermont diabetes information system. BMC endocrine disorders, 817. https://doi.org/10.1186/1472-6823-8-17
Ramos-Nino, Maria E, et al. "Association of angiotensin-converting enzyme inhibitor therapy and comorbidity in diabetes: results from the Vermont diabetes information system." BMC endocrine disorders vol. 8 (2008): 17. doi: https://doi.org/10.1186/1472-6823-8-17
Ramos-Nino ME, Maclean CD, Littenberg B. Association of angiotensin-converting enzyme inhibitor therapy and comorbidity in diabetes: results from the Vermont diabetes information system. BMC Endocr Disord. 2008 Dec 05;8:17. doi: 10.1186/1472-6823-8-17. PMID: 19061507; PMCID: PMC2632632.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 453 entries